CAS: 18787-64-9; 3-Hexadecanone

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593-03-3 45206-91-5 540-08-9

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CAS号544-76-3 正十六烷 | CAS号544-63-8 肉豆蔻酸 | CAS号75-03-6 碘乙烷 | CAS号765-09-3 1-溴十三烷 | CAS号79-03-8 丙酰氯 | CAS号593-03-3 3-十六烷醇

📜Alkaline Earth Salt Of/the/ Methylsulfuric Acid 反应生成3-十六酮
参考文献:Dominant Role Of An Endothelium-Derived Hyperpolarizing Factor (Edhf)-Like Vasodilator In The Ciliary Vascular Bed Of The Bovine Isolated Perfused Eye
标题:Dominant Role Of An Endothelium-Derived Hyperpolarizing Factor (Edhf)-Like Vasodilator In The Ciliary Vascular Bed Of The Bovine Isolated Perfused Eye
摘要:以下是中文翻译: 评估了来自内皮的no,前列环素和内皮源性超极化因子(edhf)在介导乙酰胆碱和缓激肽引发的血管舒张反应中的作用,实验对象为牛孤立灌注眼的睫状血管床. 乙酰胆碱或缓激肽引起的血管舒张不受no合成酶抑制剂(l-Name,100 μm)或环氧化酶抑制剂(flurbiprofen,30 μm)的影响,但在给予高浓度kcl(30 Mm)或使用洗涤剂chaps(0.3%,2 Min)破坏内皮后,血管舒张几乎完全消失. 乙酰胆碱诱导的血管舒张不受atp敏感型k+通道抑制剂(glibenclamide,10 μm)的影响,但在给予非选择性k+通道抑制剂(tea,10 Mm)后显著减弱. 小导电钙敏感型k+通道(sk+ca)抑制剂(apamin,100 Nm)和大导电钙敏感型k+通道(bk+ca)抑制剂(iberiotoxin,50 Nm)对乙酰胆碱诱导的血管舒张无显著影响.相比之下,中/大导电钙敏感型k+通道抑制剂(charybdotoxin,50 Nm)强烈阻断了这些血管舒张反应,并揭示了血管收缩反应. Apamin(100 Nm)与sub-Threshold浓度的charybdotoxin(10 Nm)组合显著减弱乙酰胆碱诱导的血管舒张,而apamin(100 Nm)与iberiotoxin(50 Nm)组合无影响. 综上所述,高浓度kcl,Charybdotoxin或apamin与sub-Threshold浓度charybdotoxin的组合对血管舒张的强烈阻断,强烈表明牛孤立灌注眼中的血管舒张是由edhf介导的. British Journal Of Pharmacology (2001) 134,912-920; Doi:10.1038/sj.Bjp.0704332
Doi:10.1038/sj.Bjp.0704332

专利信息

专利号:US-9388345-B2
优先权日:2012-07-03
标题:Hydrocarbon synthesis methods, apparatus, and systems
发明人：MCNEFF CLAYTON V; MCNEFF LARRY C; NOWLAN DANIEL THOMAS; YAN BINGWEN; GREUEL PETER G
权利人:SARTEC CORP
摘要:Embodiments of the invention include apparatus and systems for hydrocarbon synthesis and methods regarding the same. In an embodiment, the invention includes a process for creating a hydrocarbon product stream comprising reacting a reaction mixture in the presence of a catalyst inside of a reaction vessel to form a product mixture, the reaction mixture comprising a carbon source and water. The temperature inside the reaction vessel can be between 450 degrees Celsius and 600 degrees Celsius and the pressure inside the reaction vessel can be above supercritical pressure for water. In an embodiment, the invention includes an extrusion reactor system for creating a hydrocarbon product stream. The temperature inside the extrusion reactor housing between 450 degrees Celsius and 600 degrees Celsius. Pressure inside the reaction vessel can be above supercritical pressure for water. Other embodiments are also included herein.

专利号:EP-3004036-A1
优先权日:2013-05-31
标题 :Methods of synthesis of ingenol and intermediates thereof

专利号:US-2016107977-A1
优先权日:2013-05-31
标题 :Methods of synthesis of ingenol and intermediates thereof

专利号:WO-2014191457-A1
优先权日:2013-05-31
标题:Methods of synthesis of ingenol and intermediates thereof

专利号:EP-3004036-B1
优先权日:2013-05-31
标 题 :Methods of synthesis of ingenol and intermediates thereof
发明人：BARAN PHILLIPE S; JØRGENSEN LARS; KUTTRUFF CHRISTIAN A; MCKERRALL STEVEN J; YEH CHIEN-HUNG
权利人:LEO LABORATORIES LTD

专利号:US-7071341-B2
优先权日:2003-02-28
标 题 :α-Glucosidase inhibitors and their synthesis from a natural source
发明人：RAO JANASWAMY MADHUSUDANA; RAO RAO JAGADEESHWAR; KUMAR UPPARAPALLI SAMPATH; REDDY SINGIREDDY VENKAT; TIWARI ASHOK KUMAR; YADAV JHILLUSINGH; RAGHAVAN KONDAPURAM VIJAYA
权利人:COUNCIL SCIENT IND RES
摘要:The present invention provides novel α-glucosidase inhibitory compound (−)-mesquitol and its analogs isolated in significant yield from traditional medicinal plant Dichrostachys cinerea and further modification of (−)-mesquitol to enhance the α-glucosidase inhibitory potential; the invention also identifies the usage of (−)-mesquitol and its analogues, based on their α-glucosidase inhibitory activity, as broad based potential therapeutics as antihyperglycernic, antidiabatic, antiobesity, antiviral, anticancer, immunestimulants and the like.